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SCFA metabolites, such as acetate, propionate, and butyrate, can act on fatty acid receptors in epithelial, enteroendocrine, and neuronal cells, and they could affect the central nervous system in experimental model organisms (53, 54)

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Therefore, this review meticulously examines the contribution of mitochondrial dysfunction to DE, delves into the mechanisms by which various etiological factors induce mitochondrial dysfunction leading to DE, and synthesizes current insights into the treatment of mitochondrial dysfunction in DE