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Some research suggests that adequate vitamin B12 status may support normal metabolic function

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It has been found to inhibit glucose-6-phosphate dehydrogenase and the pentose phosphate pathway in human and mouse endometrial stromal cells and human breast cancer cells (206, 207), and to increase oxidative phosphorylation in cumulus cells of aged infertile patients and in liver mitochondria of developing rats (208, 209)