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Pharmacological depletion of GSH similarly sensitizes neurons to ferroptosis [31], demonstrating that GSH loss, whether from oxidative consumption or impaired synthesis, compromises the GSH-GPX4 antioxidant axis
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Inhibition of AR was found to prevent the growth of HT29, SW480, and Caco-2 colon cancer cells and nude mice xenografts by downregulating miR-21 expression with concomitant upregulation of programmed cell death 4 (PDCD4), a target of miR-21 by modulation of the ROS/AMPK/mTOR/AP1/4E-BP1 pathway [142]
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