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Mechanisms of methionine dependence in cancer: oxidative stress derails methionine recycling Rapidly proliferating cancer cells have an increased demand for methionine for protein synthesis, for maintenance and de novo methylation of DNA and chromatin, which requires SAM, and for the methionine cycle to generate intermediates and cofactors used in glutathione and nucleotide biosynthesis [2]
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However, the increase in D-penetratin concentration reduced nasal absorption of insulin (Khafagy et al., 2009a)
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