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For example, one study showed that deletion of NOD-like receptor X1 ( NLRX1 ) in mice can protect western diet-induced hepatic steatosis, fibrosis, obesity, insulin resistance, and glycosuria by decreasing glycolysis and increasing fatty acid oxidation in hepatocytes [156]

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Figure 5 Although this review delves into the critical role of SLC7A11 in diabetic wound healing, it must be acknowledged that diabetic wound healing is a multifactorial-driven complex process involving various aspects, such as angiogenesis, microbial infection, and diabetes-induced systemic complications