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One of the considered mechanisms of migraine pathogenesis concerns the involvement of tumor necrosis factor α (TNF-α), which results in the occurrence of migraine symptoms through initiation of neuronal hyper-excitability, stimulation of nociceptors, and prostanoid production which, in turn, leads to initiation of neuroinflammation [69]
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Figure 6 Discussion In this study, we found that co-supplementation of GSH with SeNPs has a synergistic protective effect, which is manifested up-regulation of semen motility, viability, mitochondrial activity, plasma membrane integrity, acrosome integrity, antioxidant penitential, and embryonic development