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Taken together, the authors reported strong evidence for both migration and long-term deposition of tattoo pigment, alongside conformational alteration of biomolecules which may cause cutaneous inflammation and other adverse events because of tattooing
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Phosphorylated FDX1 promoted glycolysis, as evidenced by increased pyruvate and lactate production, enhanced extracellular acidification rates (ECAR), and upregulation of glycolytic enzymes such as GLUT1, HK2, PKM2, and LDHA
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