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In both MCAO rat models and in vitro OGD/R neuron models, Ginkgolide K exerts neuroprotective effects against ischemic stroke by inhibiting GSK-3β-mediated mitochondrial hyper-fission and membrane permeability increases, thereby reducing cerebral infarct volume and neuronal apoptosis ( 3.4 Neuroprotection Dysfunction of the BBB compromises nutrient delivery to neural cells, subsequently precipitating neuronal apoptosis, synaptic impairment, and diminished neuroplasticity—processes that constitute critical mechanisms underlying cognitive decline in VCI
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