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In vitro and animal-based studies have explored AOD-9604's interactions with osteoblast and osteoclast activity, investigating its role in bone remodeling pathways independent of IGF-1 signaling

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In the “other” group, we included patients with diseases, which did not exceed three cases, such as autoimmune and fulminant hepatitis, Von Gierke disease, portal vein developmental anomaly, Crigler-Najjar syndrome, Budd-Chiari syndrome, hepatoblastoma, alpha-1 antitrypsin deficiency, and previous liver transplant dysfunction (Table 1)