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Mitochondrial-specific omics, such as mtDNA sequencing, mtDNA copy number/heteroplasmy analysis, and mitochondrial proteomics, can directly assess mitochondrial genome damage, respiratory chain complex abnormalities, and alterations in mitochondrial protein homeostasis

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A study of Korean adults found that MOTS-C levels were lower in men with metabolic syndrome and that the degree of depression in MOTS-C correlated with the number of metabolic syndrome components present