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Liu et al (139) demonstrated that miR-193a-5p can target GPX4, and that inhibition of miR-193a-5p or overexpression of GPX4 can reduce lipid ROS and Fe2+ levels in cervical cancer cells, thereby effectively inhibiting ferroptosis

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Recently, Wang et al (107) discovered that effector T cells can use inosine as an alternative substrate to support cell growth and function in the absence of glucose in vitro